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This is why patience and structured physiotherapy matter far more than any single intervention

Figure 3 A comprehensive understanding of these mechanisms in TBI is essential for expanding our knowledge of its pathological progression, identifying effective therapeutic targets, and formulating preventive strategies

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Mass Spectrom Rev 33(4):237276

Neutrophils are signaled to the areas of inflammation even without infection present, but bacterial presence can push this neutrophilic response further, generating more oxidative stress from released reactive oxygen species, more crosslinking of the mucins from the oxidative stress, and more DNA released from dying epithelial and neutrophil cells (Yuan et al., 2015

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